愛(ài)華網(wǎng)對(duì)草甘膦現(xiàn)有的批準(zhǔn)有什么錯(cuò)誤？12.1公開(kāi)發(fā)表的經(jīng)同行審查的科學(xué)文獻(xiàn)被拒絕：草甘膦除草劑致癌性。2002年的審查聲稱對(duì)草甘膦與草甘膦除草劑對(duì)致癌性“沒(méi)有證據(jù)”。但是，2002年審查以前很長(zhǎng)時(shí)間就知道草甘膦有致癌性效應(yīng)。本文系《草甘膦除草劑與生育缺陷–是否向公眾掩蓋真相？》第12章前部分。
What’s wrong with the current approval of glyphosate?:12.1 Open peer reviewed scientific literature is denied:Reveals Glyphosate/Roundup causeCarcinogenicity。The 2002 review claims“no evidence” of carcinogenicity for glyphosate andglyphosate trimesium. But glyphosate was known to have carcinogeniceffects long before the 2002 review. This paper is1st part of section 12 of “Roundup and birth defects --Is the public being kept in the dark?”
12. What’s wrong with the current approval of glyphosate?(3)
12. 對(duì)草甘膦現(xiàn)有的批準(zhǔn)有什么錯(cuò)誤？（3）
-- Roundup and birth defects-- Is the public being kept in thedark?
-- 草甘膦除草劑與生育缺陷 –是否向公眾掩蓋真相？
譯者：陳一文（cheniwan@cei.gov.cn）
Translated by Chen I-wan
《轉(zhuǎn)基因技術(shù)與人類安全》研究專家、80年代前全國(guó)青聯(lián)委員
“GM Technology & MankindSafety” researcher
《新浪網(wǎng)》“陳一文顧問(wèn)博客”：http://blog.sina.com.cn/cheniwan
譯自《地球開(kāi)放式資源》網(wǎng)站下載pdf文件：
Translated from pdf document downloadat:
http://earthopensource.org/files/pdfs/Roundup-and-birth-defects/RoundupandBirthDefectsv5.pdf
[Translator’s note: This is 1st draftof direct translation, unproof read. If errors are found, thetranslator most appreciates being notifying.]
【譯者注；本文系直譯第1稿，未經(jīng)校對(duì)。如果發(fā)現(xiàn)譯誤，感謝告知譯者?！?/p>
Carcinogenicity
致癌性
The 2002 review claims“no evidence” of carcinogenicity for glyphosate andglyphosate trimesium. But glyphosate was known to have carcinogeniceffects long before the 2002 review.
2002年的審查聲稱對(duì)草甘膦與草甘膦除草劑對(duì)致癌性“沒(méi)有證據(jù)”。但是，2002年審查以前很長(zhǎng)時(shí)間就知道草甘膦有致癌性效應(yīng)。
Two long-term studies on rats wereconducted in 1979–1981 and 1988–1990.[274] The rats received 3, 10 and 32 mg/kgof glyphosate per day in the first study and 100, 410 and 1060mg/kg per day in the second. The first study found a significantincrease in tumours in the testes of rats fed glyphosate, but thesame effect was not found in the second test using the higherdoses. On this basis, glyphosate was excluded from the carcinogeniccategory.[275, 276]
1979-1981年與1988-1990年對(duì)老鼠進(jìn)行了兩項(xiàng)長(zhǎng)期研究。[274] 第一項(xiàng)研究中，老鼠每天接受3、10與32mg/kg草甘膦，第二項(xiàng)研究中每天接受100、410與1060mg/kg草甘膦。第一項(xiàng)研究在喂食了草甘膦的老鼠試驗(yàn)中發(fā)現(xiàn)腫瘤顯著增多，但是喂食更高劑量草甘膦的第二項(xiàng)試驗(yàn)沒(méi)有發(fā)現(xiàn)同樣的效應(yīng)。在此基礎(chǔ)上，將草甘膦從致癌性類別中取消。[275、276]
274. WHO (World Health Organization).1994. Glyphosate. Environmental Health Criteria. 159. 274. WHO（世界衛(wèi)生組織）。1994。草甘膦。環(huán)境健康標(biāo)準(zhǔn)。159. http://www.inchem.org/documents/ehc/ehc/ehc159.htm#SectionNumber:7.3 275. WHO (World Health Organization).1994. Glyphosate. Environmental Health Criteria. 159. 275. 274. WHO（世界衛(wèi)生組織）。1994。草甘膦。環(huán)境健康標(biāo)準(zhǔn)。159. http://www.inchem.org/documents/ehc/ehc/ehc159.htm#SectionNumber:7.3 276. Dallegrave, E., Mantese, F. D.et al. 2003. The teratogenic potential of the herbicideglyphosate-Roundup in Wistar rats. Toxicol Lett 142(1–2):45-52. 276. Dallegrave, E., Mantese, F.D.等，2003。草甘膦除草劑農(nóng)達(dá)在Wistar鼠中的致畸形潛力。毒理學(xué)通訊，142(1–2): 45-52. |
This move was based on outdated andincorrect assumptions about toxicology. It used to be thought thattoxic effects increased in proportion to dose, and that there is asafe level of a chemical, below which toxic effects are not found.But toxicologists now know that these assumptions are not alwaystrue. Some chemicals have more potent effects (notably endocrineeffects) at l ow doses than higher doses.[277] In some cases, no safe threshold canbe found.[278, 279] However, regulators have not revisedtheir conclusions on glyphosate based on up-to-date scientificknowledge.
這樣一項(xiàng)措施基于對(duì)于毒理學(xué)的過(guò)時(shí)了的以及不正確的假設(shè)。原本想像毒性效應(yīng)隨劑量按比例增加，而且認(rèn)為對(duì)一種化學(xué)品存在著一個(gè)安全水平，毒性效應(yīng)在這個(gè)水平之下不被發(fā)現(xiàn)。但是，毒理學(xué)家們現(xiàn)在知道，這些假設(shè)不一定總正確。某些化學(xué)品在低劑量比高劑量具有威力更強(qiáng)大的效應(yīng)（特別是內(nèi)分泌效應(yīng)）。[277] 在某些情況下，找不到它們的安全門檻（安全閾值）。[278、279] 然而，政府監(jiān)管者們沒(méi)有基于最新的科學(xué)知識(shí)修正他們對(duì)草甘膦的結(jié)論。
277. Gierthy, J. F. 2002. Testing forendocrine disruption: how much is enough? Toxicol Sci 68(1):1-3. 277. Gierthy, J.F.，2002。對(duì)內(nèi)分泌干擾進(jìn)行試驗(yàn)：多少足夠？毒理學(xué)科學(xué)，68(1): 1-3. 278. Sheehan, D. M. 2006.No-threshold dose-response curves for nongenotoxic chemicals:findings and applications for risk assessment. Environ Res 100(1):93-99. 278. Sheehan, D.M.，2006。對(duì)非致基因毒性化學(xué)品沒(méi)有閾值劑量-反應(yīng)曲線：對(duì)風(fēng)險(xiǎn)評(píng)估的發(fā)現(xiàn)與應(yīng)用。環(huán)境研究，100(1): 93-99.  279. Vom Saal, F. S. and Hughes,C. 2005. An extensive new literatureconcerning low-dose effects of bisphenol A shows the need for a newrisk assessment. Environmental Health Perspectives 113:926–933. 279. Vom Saal, F. S. and Hughes,C.，2005。有關(guān)雙酚A（BPA）低劑量影響的廣泛新的文獻(xiàn)表明進(jìn)行新的風(fēng)險(xiǎn)評(píng)估的需要。環(huán)境性健康前景，113: 926–933. |
Studies from the independentliterature also show that Roundup and glyphosate have carcinogeniceffects:
獨(dú)立文獻(xiàn)的研究表明草甘膦除草劑農(nóng)達(dá)與草甘膦具有致癌性效應(yīng)：
●Glyphosate induces cancer in mouseskin[280]
●草甘膦在小鼠皮膚中誘發(fā)癌[280]
●Epidemiological studies show a linkbetween Roundup/glyphosate exposure and two types of cancer:multiple myeloma[281] and non-Hodgkin’s lymphoma.[282, 283, 284]
●流行病學(xué)研究表明草甘膦除草劑農(nóng)達(dá)/草甘膦暴露與兩種癌癥關(guān)聯(lián)：多發(fā)性骨髓瘤[281]與非霍奇金淋巴瘤。[282、283、284]
●Other studies (mentioned underGenotoxicity, above) show that Roundup, glyphosate, and itsmetabolite AMPA cause changes to cells and DNA that are known tolead to cancer.[285, 286, 287, 288, 289, 290]
●其他的研究（在前述“致基因毒性”一節(jié)中提到）表明，草甘膦除草劑農(nóng)達(dá)、草甘膦、及其代謝物AMPA對(duì)細(xì)胞與DNA造成已知導(dǎo)致癌癥的變化。[285、286、287、288、289、290]
280. George, J., Prasad, S., Mahmood,Z., Shukla, Y. 2010. Studies on glyphosate-induced carcinogenicityin mouse skin: A proteomic approach. J Proteomics 73:951–964. 280. George, J., Prasad, S., Mahmood,Z., Shukla, Y.，2010。對(duì)小鼠皮膚中草甘膦誘發(fā)致癌性的研究：一種蛋白質(zhì)組學(xué)方法。蛋白質(zhì)組學(xué)雜志，73: 951–964. 281. De Roos, A. J., Blair, A.,Rusiecki, J. A., et al. 2005. Cancer incidence amongglyphosate-exposed pesticide applicators in the Agricultural HealthStudy. Environ Health Perspect. 113(1): 49–54. 281. De Roos, A. J., Blair, A.,Rusiecki, J. A.等，2005。農(nóng)業(yè)健康研究中草甘膦暴露農(nóng)藥噴灑者中的癌癥發(fā)生率。環(huán)境性健康前景，113(1): 49–54. 282. Hardell, L., Eriksson, M. 1999.A case-control study of non-Hodgkin lymphoma and exposure topesticides. Cancer. 85(6): 1353–1360. 282. Hardell, L., Eriksson,M.，1999。非霍奇金淋巴瘤與農(nóng)藥暴露的對(duì)照組研究。癌癥。85(6): 1353–1360. 283. Hardell, L., Eriksson, M., Nordstrom,M. 2002. Exposure to pesticides as risk factorfor non-Hodgkin’s lymphoma and hairy cell leukemia: pooled analysisof two Swedish case-control studies. Leuk Lymphoma. 43(5): 1043–1049. 283. Hardell, L., Eriksson, M., Nordstrom,M.，2002。農(nóng)藥暴露作為對(duì)非霍奇金淋巴瘤與多毛細(xì)胞白血病的風(fēng)險(xiǎn)因素：兩項(xiàng)瑞典對(duì)照組研究的合并分析。白血病淋巴瘤。43(5): 1043–1049. 284. Eriksson, M., Hardell, L.,Carlberg, M., Akerman, M. 2008. Pesticide exposure as risk factorfor non-Hodgkin lymphoma including histopathological subgroupanalysis. Int J Cancer. Oct 1 2008;123(7): 1657–1663. 284. Eriksson, M., Hardell, L.,Carlberg, M., Akerman, M.，2008。農(nóng)藥暴露作為對(duì)非霍奇金淋巴瘤的風(fēng)險(xiǎn)因素，包括組織病理學(xué)子群分析。國(guó)際癌癥雜志，10月1日，2008;123(7): 1657–1663. 285. Mañas, F., Peralta, L., Raviolo,J., Garcia, O.H., Weyers, A., Ugnia, L., Gonzalez, C.M., Larripa,I., Gorla, N. 2009. Genotoxicity of glyphosate assessed by theComet assay and cytogenic tests. Environ Toxicol Pharmacol 28:37–41. 285. Mañas, F., Peralta, L., Raviolo,J., Garcia, O.H., Weyers, A., Ugnia, L., Gonzalez, C.M., Larripa,I., Gorla, N.，2009。由彗星分析與細(xì)胞基因試驗(yàn)評(píng)估的草甘膦致基因毒性。環(huán)境毒理學(xué)藥理學(xué)，28: 37–41. 286. Manas, F., Peralta, L. et al.2009. Genotoxicity of AMPA, the environmental metabolite ofglyphosate, assessed by the Comet assay and cytogenetic tests.Ecotoxicol Environ Saf 72(3): 834–837. 286. Manas, F., Peralta,L.等，2009。草甘膦環(huán)境代謝物AMPA由彗星分析與細(xì)胞基因試驗(yàn)評(píng)估的致基因毒性。生態(tài)毒性環(huán)境安全，72(3): 834–837. 287. Marc, J., Mulner-Lorillon, O.,Belle, R. 2004. Glyphosate-based pesticides affect cell cycleregulation. Biol Cell. 96(3): 245–249. 287. Marc, J., Mulner-Lorillon, O.,Belle, R.，2004。草甘膦為基礎(chǔ)農(nóng)藥影響細(xì)胞周期雕制。生物細(xì)胞。96(3): 245–249. 288. Bellé, R., Le Bouffant, R., Morales, J.,Cosson, B., Cormier, P., Mulner-Lorillon O. 2007. Sea urchinembryo, DNA-damaged cell cycle checkpoint and the mechanismsinitiating cancer development. J Soc Biol. 201: 317–327 288. Bellé, R., Le Bouffant, R., Morales, J.,Cosson, B., Cormier, P., Mulner-Lorillon O.，2007。海膽胚胎，DNA損傷細(xì)胞周期檢查點(diǎn)與啟動(dòng)癌癥發(fā)展的機(jī)制。社會(huì)生物學(xué)雜志，201: 317–327 289. Marc, J., Mulner-Lorillon, O.,Boulben, S., Hureau, D., Durand, G., Bellé, R. 2002. Pesticide Roundup provokescell division dysfunction at the level of CDK1/cyclin B activation.Chem Res Toxicol. 15(3): 326–331. 289. Marc, J., Mulner-Lorillon, O.,Boulben, S., Hureau, D., Durand, G., Bellé, R.，2002。草甘膦除草劑農(nóng)達(dá)在CDK1/細(xì)胞周期蛋白活化水平激發(fā)細(xì)胞分類功能失調(diào)?；瘜W(xué)研究毒理學(xué)。15(3): 326–331. 290. Marc, J.,Bellé, R., Morales, J., Cormier, P.,Mulner-Lorillon, O. 2004. Formulated glyphosate activates theDNA-response checkpoint of the cell cycle leading to the preventionof G2/M transition. Toxicol Sci. 82(2): 436–442. 290. Marc, J.,Bellé, R., Morales, J., Cormier, P.,Mulner-Lorillon, O.，2004。配方的草甘膦啟動(dòng)細(xì)胞周期DNA-反應(yīng)檢查點(diǎn)導(dǎo)致防止G2/M傳遞。毒理學(xué)科學(xué)。82(2): 436–442. |
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必須追究農(nóng)業(yè)部非法審批孟山都轉(zhuǎn)基因大豆進(jìn)口的法律責(zé)任
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